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The pathophysiology of sepsis and mechanisms of multiple organ system dysfunction are reviewed here A review of the literature and rationale for further research The definition and management of sepsis are discussed separately.
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The pathophysiology of sepsis is complex and results from the effects of circulating bacterial products, mediated by cytokine release, caused by sustained bacteraemia Lactate kinetics in sepsis and septic shock Cytokines are primarily responsible for the clinically observable effects of the bacteraemia in the host.
In its most severe form, sepsis causes multiple organ dysfunction that can produce a state of chronic critical illness characterized by severe immune dysfunction and catabolism
Much has been learnt about the pathogenesis of sepsis at the molecular, cell, and intact organ level. Sepsis represents a critical condition marked by systemic inflammation, widespread tissue damage, and organ failure, which can lead to significant morbidity and mortality The pathophysiology of sepsis involves a complex interplay between the immune system and pathogenic microorganisms. The complement system is integral to the pathophysiology of sepsis, a critical and frequently lethal disorder defined by an excessive immune reaction to infectious agents.
Today, the heterogeneous syndrome is defined as severe organ dysfunction caused by a dysregulated host response to infection, with renewed emphasis on immune pathophysiology. Immune cell activation against the antigens causes severe distress that mediates a strong inflammatory response in vital organs. Chertoff j, chisum m, garcia b, lascano j
